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Mechanisms Underlying the Contribution of Type 1 Diabetes Risk Associated Variants (DP3)

Post Date

July 7th 2015

Application Due Date

February 17th 2016

Funding Opportunity Number

RFA-DK-15-025

CFDA Number(s)

93.847
93.855
93.856

Funding Instrument Type(s)

Grant

Funding Activity Categories

Food and Nutrition
Health

Eligibility Categories

State Governments
County Governments
City or Township Governments
Special District Governments
Independent School Districts
Public and State Controlled Institutions of Higher Education
Federally Recognized Native American Tribal Governments
Public Housing Authorities or Indian Housing Authorities
Non-Federally Recognized Native American Tribal Organizations
Non-Profits With 501 (c) (3) Status With The IRS (Except Higher Education Institutions)
Non-Profits Without 501 (c) (3) Status With The IRS (Except Higher Education Institutions)
Private Institutions of Higher Education
For-Profit Organizations (Except Small Businesses)
Small Businesses
Other

Other Eligible Applicants include the following: Alaska Native and Native Hawaiian Serving Institutions; Asian American Native American Pacific Islander Serving Institutions (AANAPISISs); Eligible Agencies of the Federal Government; Faith-based or Community-based Organizations; Hispanic-serving Institutions; Historically Black Colleges and Universities (HBCUs); Indian/Native American Tribal Governments (Other than Federally Recognized); Non-domestic (non-U.S.) Entities (Foreign Organizations); Regional Organizations; Tribally Controlled Colleges and Universities (TCCUs) ; U.S. Territory or Possession.

Funding

  • Estimated Total Funding:

    $15000000

  • Award Range:

    $None - $600000

Grant Description

This Funding Opportunity Announcement (FOA) encourages research project grantapplications from institutions/organizations for projects to characterize the genetic variations in human genomic regions that have been putatively associated with type 1 diabetes (T1D) and conduct follow-up functional studies of particular genetic variants. In addition there are many human T1D regions for which there is no compelling functional candidate gene and thus additional work to identify causal genes and potential causal variants and elucidate the mechanisms whereby changes in the function or regulation of these genes are likely to provide crucial new insights into disease pathogenesis are also encouraged.

Contact Information


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